According to a new study, the retina of the human eye is not different from those of all vertebrates, including the dog and mouse.
That’s good news for people with retinal disease.
Retinal diseases affect one in every three people, with over 4 million people diagnosed worldwide, according to the World Health Organization.
This is why retinal research is so important for vision researchers and other scientists who are studying the human retina.
Retina disease affects vision in a number of ways, including photoreceptor degeneration, or the loss of photoreceptors, which make up our visual field.
The retina is a special kind of lens that covers the eye.
As it ages, this lens loses some of its natural shape, so it becomes less flexible and therefore more susceptible to damage from UV radiation.
As a result, the amount of light that can reach the retina is reduced, and it starts to become cloudy and less clear.
Retinas are usually found on the back of the eye and are surrounded by an outer layer of tissue called the macula, which acts as a protective covering.
Retinitis pigmentosa (RP) is a very rare disorder in which the macular epithelium (the layer of fat that covers and protects the retina) fails to form and is unable to absorb any light, making it hard for the retinal pigment epithelia to produce the proper signals for vision.
In many people, the maculopathy doesn’t appear until late in life, when the body has aged for a long time and has developed more damage to the retina.
As such, people with RP often have difficulty seeing in dark environments.
In a study published in 2016, researchers from the University of Utah showed that, as a result of their study, a group of volunteers were able to improve their vision in the dark using a combination of retinal regeneration and retinal photoreduction therapy.
They also showed that the improvement was related to the amount and types of retinas in their eye.
Now, a new group of researchers at the University at Buffalo has shown that the same retinas are found in people with vision loss due to RP, even though they had no problems with vision in their eyes at the beginning of their recovery.
“The macular pigment epithelial system (MPEs) and retinoids that form the outermost layer of the macaque retina do not regenerate,” lead author James D. Tait told the New York Times.
“So, these cells are not getting the right signals to regenerate properly.”
In other words, RP patients are missing out on their natural vision, and as a consequence, their vision suffers.
In this new study published online in Nature Medicine, Tait and his team looked at the retinas of 16 people with severe RP who underwent retinal retinal transplantation, and found that while all 16 people had normal visual acuity (VA) and visual acuteness (VA/VAE), one person had a significant loss of vision that was completely irreversible.
The researchers compared that person’s vision with that of a normal person, who also underwent a retinal reconstruction.
“What surprised us was that we actually didn’t see any difference in the person with RP versus the person without RP,” said Tait.
“This person had all these amazing results, but they weren’t improving.
The person without the retina loss had no improvement at all.”
In a follow-up study, Tatsait and another group of people with the same diagnosis were able, in a small number of cases, to see improvements in vision in those patients with RP.
However, those results weren’t statistically significant.
The new study has important implications for how the human body uses retinal cells, and how we treat vision loss with treatments that increase vision.
“If we can learn something from the retinitis pigments in the retina, we can probably find a way to improve vision and repair damage in the eyes,” Tait said.
“We have a great deal of potential in the human brain to repair damage that we’ve seen in other animals and the human immune system.”
The researchers are now working on further studies to test how retinal restoration improves vision in people who don’t have vision loss.